目的：探讨丘脑在听觉Ｐ３产生中的作用。方法：对丘脑出血恢复期患者进行两次事件相关电位、脑ＣＴ和简易精神状态检查。结果：丘脑出血患者Ｐ３波幅正常，但Ｐ３峰潜伏期延长．Ｐ３ＰＬ延长并非颅内高压和认知损害引起。结论：丘脑的多个核团参与Ｐ３的发生，丘脑在听觉Ｐ３产生过程中起触发性作用。 Objective: To explore the role of the thalamus in the production of auditory P3. Methods: Two episodes of event-related potentials, brain CT and brief mental state were performed in patients with convalescent thalamic hemorrhage. Results: The amplitude of P3 in patients with thalamic hemorrhage was normal, but the latency of P3 peak was prolonged. P3PL prolongation is not caused by intracranial hypertension and cognitive impairment. CONCLUSIONS: Multiple nuclei of the thalamus are involved in the development of P3, and the thalamus plays a triggering role in the production of auditory P3.