用血浆蛙膀胱囊-压力换能器法,在人工呼吸制动下,电刺激18只家兔下丘脑内侧区一定部位,记录胆囊压力,在10～15秒潜伏期后,压力下降,刺激20～30秒后达到最低值,比基础压低1～3厘米水柱,停刺后压力回升,3分钟后恢复至对照水平。胆囊压力下降不受阿托品影响,受心得安抑制,这提示其机制可能是通过交感神经β受体而实现的。 Under the action of artificial respiration, 18 rabbits were electrically stimulated to a certain site in the medial hypothalamic area to record the pressure of the gallbladder. After a latencies of 10-15 seconds, the pressure was decreased and the stimulus was 20 ~ After 30 seconds to reach the lowest value, lower than the base of 1 to 3 cm water column, pressure rose after stopping stab, 3 minutes later returned to the control level. The decrease of gallbladder pressure was not affected by atropine, and was suppressed by heart, suggesting that the mechanism may be achieved by the sympathetic β receptors.