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近年来,许多用细胞培养的研究都表明,CAMP对自发转化和病毒转化的细胞均有抑制作用,为了探讨在活体动物体内,cAMP与肿瘤发生和发展的关系,作者测定了化学致癌物二乙基亚硝胺诱发成恶性肿瘤后,肿瘤小鼠血浆、瘤纰织、瘤周组织和接种肉瘤180小鼠血浆cAMP的含量,并和正常同种动物的测定值进行了比较。血浆测定的结果;二乙基亚硝胺诱发的肿瘤鼠血浆cAMP显著地高亍正常鼠血浆的含量,而接种肉瘤180鼠血浆cAMP水平与正常鼠没有明显的差别。组织测定的结果是:肺瘤组
In recent years, many studies using cell cultures have shown that CAMP inhibits both spontaneous transformation and viral transformation. To investigate the relationship between cAMP and tumorigenesis and development in living animals, the authors determined chemical carcinogens. After the nitrosamine-induced malignant tumors, plasma cAMP levels in plasma, neoplasm, tumor tissue, and sarcoma-inoculated 180 mice were compared with those of normal animals of the same species. As a result of the plasma assay, diethylnitrosamine-induced tumor cAMP plasma levels were significantly higher in normal rat plasma than in normal mice, whereas plasma cAMP levels in inoculated sarcoma 180 mice were not significantly different from normal mice. The result of the tissue test is: lung tumor group