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目的 :观察胞肌对视神经萎缩的治疗修复作用。方法 :视神经萎缩病人 60例 (男性 4 9例 ,女性 11例 ,38a±s14a)随机分为 2组 (每组 30例 ) ,胞肌组给予注射用胞肌 30 0mg ,妥拉唑林组给予妥拉唑林 2 5mg ,均球后注射给药 ,qod× 6wk。分别测定用药前后的视力、视野、视诱发电位 (VEP)。结果 :胞肌组的总有效率 70 % ,妥拉唑林组为 13% ,差异有非常显著意义 (P <0 .0 1) ;治疗后临床各项指标的恢复率为视力 >视野 >VEP ;对各种原因引起视神经萎缩的疗效以视网膜色素变性所致的视神经萎缩有效率最高 ( 86% ) ,其次是视神经炎后视神经萎缩 ( 53% ) ,遗传性的视神经萎缩效果较差 ,青光眼和钝挫伤引起视神经萎缩因病例太少未予分析。结论 :胞肌是一种有效治疗视神经萎缩的药物 ,无明显不良反应
Objective: To observe the repair and treatment of optic nerve atrophy by cytoplasm. Methods: 60 cases of optic atrophy (49 males and 11 females, 38a ± s14a) were randomly divided into 2 groups (30 in each group) Toxoprofen 2 5mg, after the ball injection, qod × 6wk. Visual acuity, visual field and visual evoked potential (VEP) were measured before and after treatment. Results: The total effective rate was 70% in the muscle cell group and 13% in the tolazoline group, the difference was significant (P <0.01). The recovery rate of all indexes after treatment was visual acuity> visual field> VEP ; For a variety of causes of optic atrophy efficacy of retinal pigment degeneration caused by optic atrophy is the highest efficiency (86%), followed by optic neuritis optic atrophy (53%), hereditary optic atrophy is poor, glaucoma and Blunt contusion caused optic atrophy due to too few cases were not analyzed. Conclusion: The muscle cell is an effective treatment of optic atrophy drugs, no obvious adverse reactions