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目的对孕妇胎膜早破合并羊膜腔感染综合征的临床特点及诊断方法进行临床分析。方法观察组选取60例胎膜早破合并羊膜腔感染综合征孕妇,对照组选取同期60例胎膜早破无羊膜腔感染综合征孕妇,对比两组影响因素,孕妇临床表现,产妇及新生儿结局,并进行对症治疗。结果观察组破膜时间长于对照组,破膜后反复阴道检查次数高于对照组,破膜前有性生活史10.00%和合并阴道炎发病数20.00%,高于对照组(0%;3.33%)(P<0.05)。观察组孕妇体温>37.8℃发生率为76.67%(46/60);孕妇心率>100次/分钟发生率为35.00%(21/60);胎心率>160次/分钟发生率为30.00%(18/60);CRP>8 mg/L发生率为78.33%(47/60);末梢血白细胞>15×109/L发生率为98.33%(59/60)。观察组剖宫产、产程延长、产褥感染、产后出血明显高于对照组(P<0.05);观察组胎儿宫内窘迫、围生儿感染、围生儿死亡、新生儿窒息明显高于对照组(P<0.05)。结论胎膜早破是羊膜腔感染综合征的重要诱因,要降低羊膜腔感染综合征的发生率,应控制和规范医源性操作,注重健康教育,一旦发生胎膜早破,应积极采取有效地预防措施,避免羊膜腔感染综合征的发生。
Objective To analyze the clinical features and diagnostic methods of premature rupture of membranes and amniotic infection syndrome in pregnant women. Methods Sixty pregnant women with premature rupture of membrane and amniotic cavity infection syndrome were selected in the observation group. Sixty pregnant women with premature rupture of membranes without amniotic cavity infection syndrome were selected in the control group. The influencing factors, clinical manifestations, maternal and newborn Outcome, and symptomatic treatment. Results The time of rupture of the observation group was longer than that of the control group. The number of repeated vaginal examination after rupture of membranes was higher than that of the control group. The history of sexual life before the rupture of membranes was 10.00% and the incidence of vaginitis was 20.00%, higher than that of the control group (0%; 3.33% ) (P <0.05). In the observation group, the incidence rate of body temperature> 37.8 ℃ was 76.67% (46/60) in pregnant women and 35.00% (21/60) in pregnant women with heart rate> 100 beats / min. The incidence of fetal heart rate> 160 beats / minute was 30.00% 18/60). The incidence of CRP> 8 mg / L was 78.33% (47/60). The incidence of peripheral blood leukocytes> 15 × 109 / L was 98.33% (59/60). Observation group, cesarean section, prolonged labor, puerperal infection, postpartum hemorrhage was significantly higher than the control group (P <0.05); observation group fetal distress, perinatal infection, perinatal death, neonatal asphyxia was significantly higher than the control Group (P <0.05). Conclusion Premature rupture of membranes is an important inducing factor of amniotic cavity infection syndrome. To reduce the incidence of amniotic cavity infection syndrome, iatrogenic operation should be controlled and standardized, and health education should be emphasized. In the event of premature rupture of membranes, active premature rupture of membranes should be taken actively Preventive measures to prevent the occurrence of amniotic cavity infection syndrome.