论文部分内容阅读
动眼神经的迷走再生见于严重头部创伤,或后交通动脉瘤破裂所致之急性动眼神经麻痹之后。作者报导4例患者皆无早先的动眼神经麻痹经过,而有与动眼神经迷走再生相一致的,缓慢进行的眼球活动及瞳孔异常。每例均经开颅术发现有侵及海绵窦之脑膜瘤。海绵窦的任何疾病过程(肿瘤、动脉瘤、感染、炎症)均可断裂颅神经Ⅲ、Ⅳ、Ⅵ之海绵窦内段,造成由单一眼肌麻痹至眼球完全不能活动之种种眼运动障碍。
Ocular vagal regeneration occurs after severe head trauma or acute oculomotor paralysis caused by ruptured posterior communicating aneurysm. The authors reported that none of the 4 patients had previous oculomotor palsy, and had slow eye movements and pupillary abnormalities consistent with oculomotor vagal regeneration. Each case was found by invasive craniotomy and cavernous sinus meningiomas. Any disease process of the cavernous sinus (tumor, aneurysm, infection, inflammation) can break the cranial nerves Ⅲ, Ⅳ, Ⅵ of the cavernous sinus segment, resulting in a single eye muscle paralysis to the eye completely unable to move the kind of eye dyskinesia.