论文部分内容阅读
重症肌无力 (myasthenia gravis,MG)是神经肌肉接头处乙酰胆碱受体 (acetycholine receptor,Ach R)自身致敏和破坏所致的自身免疫病。其发病机制主要与抗 Ach R抗体 (anti Ach Rantibody,Ach R ab)介导的体液免疫和 T细胞介导的细胞免疫有关 ,但胸腺是激活和维持 MG自身免疫反
Myasthenia gravis (MG) is an autoimmune disease caused by the sensitization and destruction of acetycholine receptor (AchR) at the neuromuscular junction itself. Its pathogenesis is mainly related to anti-Ach R antibody (Ach Rab) -mediated humoral immunity and T cell-mediated cellular immunity, but the thymus is activated and maintained MG autoimmune anti