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既往已观察到绝经后或老年性骨质疏松引起的新鲜脊椎骨折患者摄入降钙素后血浆β-内啡肽水平升高。为了探讨β-内啡肽的增长是否为垂体源性的,是否为一种生理反应抑或仅仅与骨的病理改变相关。作者对8名51～81岁由于绝经后或老年性骨质疏松所致的新鲜脊椎骨折患者(OSt组)及7名20～40岁正常健康妇女(NC组)进行了有关给与降钙素后ACTH、β-内啡肽及可的松反应的观察。其中12名该三种激素明显的增长,摄入后15-60分钟达最高值。可的松水平(μg/dl)在st组由14.3
It has been previously observed that post-menopausal or senile osteoporosis caused by fresh vertebral fractures in patients with calcitonin plasma β-endorphin levels. To investigate whether beta-endorphin is pituitary-derived, whether it is a physiological response or is only associated with pathological changes in bone. The authors investigated the effects of calcitonin on eight patients (OSt group) and seven normal healthy women aged 20-40 years (NC group) aged 51-81 years with osteoporosis caused by postmenopausal or senile osteoporosis Post-ACTH, β-endorphin and cortisone reaction observed. Twelve of these three hormones were significantly increased, reaching their highest level 15-60 minutes after ingestion. Cortisol level (μg / dl) in st group by 14.3