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Accumulation of β-amyloid(Aβ)peptide is critical for the pathogenesis of Alzheimers disease.
TRPC6 specifically interacts with APP to inhibit its cleavage by γ-secretase and reduce Aβ productio
【摘 要】
：
Accumulation of β-amyloid(Aβ)peptide is critical for the pathogenesis of Alzheimers disease.
【机 构】
：
Laboratory of Neural Signal Transduction,Institute of Neuroscience,SIBS,State Key Laboratory of Neur
【出 处】
：
The 6th FAONS Congress and 11th Biennial Conference of CNS(第
【发表日期】
：
2015年3期
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