【摘 要】
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Objectives The purpose of this study is to assess the relation of electrocardiographic QRS duration to left ventricular myocardial scar valued by SPECT myocardial perfusion imaging in nonischemic and
【机 构】
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Jiang Su Provincial people's hospital
【出 处】
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中华医学会心电生理和起搏分会第十一次全国学术年会暨第五届扬子江心脏论坛
论文部分内容阅读
Objectives The purpose of this study is to assess the relation of electrocardiographic QRS duration to left ventricular myocardial scar valued by SPECT myocardial perfusion imaging in nonischemic and dilated cardiomyopathy patients
其他文献
室性早搏,可见于正常人和各种心脏病患者.随着对心律失常性心肌病的进一步认识,人们对室性早搏的治疗更为积极.通过体表心电图能够对室性早搏进行定位诊断、明确性质,对指导射频消融治疗有关键的作用.
预激综合征合并心房颤动是临床上常见的恶性心律失常,它除了会导致左室收缩不协调,从而引起全身血流动力学紊乱之外,也可诱发室速、室颤等致死性心律失常的发生.因此工作中快速识别预激合并房颤的心电图是十分重要的.
Purpose the of this study was to clarify the characteristic of R-wave peak time in multi-chamber ventricular pacing model,verify its value in differential diagnosis of wide QRS complex tachycardias.
目的 分析新疆地区维吾尔族和汉族人群Gs蛋白α亚基基因(GNAS1)T393C单核苷酸多态性(SNP)与非瓣膜性心房颤动(房颤)的发生是否存在相关性.方法 选取新疆地区非瓣膜性房颤患者维吾尔族103例及汉族102例,选取健康人群维吾尔族100例及汉族113例,采用聚合酶链反应-限制性内切酶片段长度多态性(PCR-RFLP)分析非瓣膜性房颤患者GNAS1基因T393C基因型及等位基因频率分布特点.
目的 了解新疆地区维吾尔族和汉族人群中白细胞介素6(IL-6)-174G/C、-572C/G、-597G/A位点基因多态性与心房颤动(房颤)的易感性是否有关.方法 选取新疆地区房颤患者维吾尔族103例及汉族102例,选取维吾尔族正常对照100例及汉族正常对照111例,采用聚合酶链反应-限制性内切酶片段长度多态性(PCR-RFLP)分析房颤患者白介素-6-174G/C、-572C/G、-597G/A
快室率的心房颤动(房颤)可影响患者心室舒张充盈功能,心房泵血功能丧失,心排出量降低,心肌缺血、心脏功能恶化,甚至出现低血压、休克等血流动力学障碍.房颤时持续增快的心室率可致心动过速性心肌病.房颤时最常见的症状,多由于快心室率和不规则心律所引起,控制心室率不仅可减轻症状,对于部分房颤患者而言心室率控制后可改善心功能.
目的 探讨肾交感神经高频电刺激(RSN-S)对缺血性室性心律失常的影响.方法 18只体重17-20Kg的正常成年杂种犬麻醉后暴露左侧肾动脉,在肾动脉表面通过高频电刺激(20Hz,2ms脉宽)定位有效的刺激靶点,并在相应靶点固定刺激电极.
Background Dilated cardiomyopathy(DCM),which is characterized by left ventricular enlargement,systolic dysfunction and heart failure.Many cases(20%–35%)of DCM are familial,suggests that genetic factor
Circadian rhythms influence the incidence of sudden cardiac death after chronic heart failure(CHF)but the underlying mechanisms are not well defined.We sought to investigate the role of the β-adrenerg
Background β2 adrenergic receptor(β2-AR)activation increases risk of sudden cardiac death(SCD)in heart failure(HF).Non-selective β-AR blockers have larger beneficial effects on survival than selective