论文部分内容阅读
Serum chemokine network correlates with chemotherapy in non-small cell lung cancer
【作 者】
：
【机 构】
：
Department of Pulmonary Medicine,Zhongshan Hospital,Fudan University,Shanghai,200032,China;Shanghai
【出 处】
：
中华医学会第七次全国高原医学学术会议暨中国病理生理学会第九次全国缺氧和呼吸病理生理学术会议
【发表日期】
：
2014年4期
其他文献
High-altitude cerebral hypoxia (HACE) is a fatal acute mountain sickness (AMS).Publications implicate hypoxia-elicited inflammation, but if it correlates to HACE remains unclear.We have reported that
会议
Hypoxic environment reduced oxygen content in DG and SVZ and promotes neurogenesis in adult rat brai
Minocyclineameliorates hypoxia-inducedblood-brain barrier damage by inhibition of HIF-1t through SIR
Severe hypoxia can induce the blood-brain barrier (BBB) disruption and cerebral edema, even cerebral hernia and death.Hypoxia-inducible factor-1α (HIF-1α) is one of the most important factors for the
会议
The voltage-gated K+ (Kv) channels play an essential role in the etiology of chronic hypoxic pulmonaryarterial hypertension (CH-PAH).Tanshinone ⅡA (TⅡA), a major active component of Salvia miltiorrhiz
肺损伤后肌体启动自我修复的过程，这个过程伴随着细胞的凋亡，自噬，炎症因子动态释放，纤维蛋白原的分解吸收，内皮和上皮细胞的重新修复。干细胞参与了改修复过程，包括内源性骨髓干细胞的动员，组织器官干细胞的增生，外源性干细胞的定植与分化。我们设计了一系列实验，来验证肺损伤后内皮和上皮修复的机制，尤其此过程中生长因子与干细胞的作用。在高原肺水肿模型中，应用真空加运动踏板复制海拔4700米的高原肺水肿模型。预
会议
MKL1 potentiates lung cancer cell migration and invasion by epigenetically activating MMP9 transcrip
Malignant tumors are exemplified by excessive proliferation and aggressive migration/invasion contributing to increased mortality of cancer patients.Matrix metalloproteinase 9 (MMP9) expression is pos
Activation of medullary α2-adrenoceptors in vitro does not disinhibit laryngeal motoneurons: failure
The preganglionic recurrent laryngeal motoneurons (PRLMs) dominate the neural control of almost allthe intrinsic laryngeal muscles.These neurons receive noradrenergic projection, and systemic administ
Activation of medullary α1A-adrenoceptors increases airway vagal tone via enhanced excitatory inputs
In mammals including the human beings, the neural control of airway smooth muscles is dominated by medullary airway vagal preganglionic neurons (AVPNs), especially those located in the external format