• 不 限
  • 期刊论文
  • 硕博论文
  • 会议论文
  • 报 纸
  • 英文论文
  2. 您的位置
  3. 首页
  4. 会议论文
  5. Overexpression of AIB1 in Papillary Thyroid Carcinoma Positively Correlates with Lymph Node Metastas

Overexpression of AIB1 in Papillary Thyroid Carcinoma Positively Correlates with Lymph Node Metastas

来源 :2012医学科学前沿暨第二届个体化治疗与抗肿瘤药物研究新趋向研讨会 | 被引量 : 0次 | 上传用户：tklyzh1
【摘 要】
：
Background:AIB1,a new oncogenic coactivator,is commonly overexpressed and amplified in variety of human cancers.The status of AIB1 expression in papillary thyroid carcinoma (PTC) and its clinical sign
【作 者】
：
Mu-Yuan Liu Hai-Peng Guo Chao-Qun Hong Han-Wei Peng Xi-Hong Yang Hao Zhang 
【机 构】
：
From Head and Neck Department,Cancer Hospital,Shantou University,Shantou,515000,China
【出 处】
：
2012医学科学前沿暨第二届个体化治疗与抗肿瘤药物研究新趋向研讨会
【发表日期】
：
2012年11期
下载到本地 , 更方便阅读
下载此文 赞助VIP
声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架
论文部分内容阅读
　　Background:AIB1,a new oncogenic coactivator,is commonly overexpressed and amplified in variety of human cancers.The status of AIB1 expression in papillary thyroid carcinoma (PTC) and its clinical significance are unclear.Methods:in this study,AIB1 expression in PTC was examined by immunohistochemistry using tissue microarrays comprised of 90 PTC specimens and 20 normal thyroid tissue specimens.
其他文献
良性前列腺增生伴糖尿病患者尿动力学分析的临床意义
目的：探讨糖尿病对良性前列腺增生(BPH)患者膀胱功能改变的影响。方法：回顾性分析166例前列腺增生患者的资料，根据是否合并糖尿病将患者分为研究组和对照组，比较两组临床资料和尿动力学检查结果之间关系。结果：研究组患者中的残余尿量显著高于对照组(P＜0.05)；研究组中最大膀胱压测定容积(MCC)、膀胱顺应值(BC)、膀胱感觉功能受损比例和膀胱过度活动症比例(OAB)明显高于对照组；排尿期最大逼尿肌
会议
良性前列腺增生糖尿病尿流动力学膀胱功能
Imaging in Chinese patients with erectile dysfunction
Objectives To assess the role of Color Doppler Flow Imaging (CDFI) in the evaluation of Chinese patients with erectile dysfunction (ED) and to explore the association between penile rigidity and hemo-
会议
A new granding system for male erectile dysfunction (ED) on the selection of its therapeutic modalit
Objective:To investigate a reasonable pattern of goal diagnosis and therapy for male erectile dysfunction.Materials and methods:196male patients suffered with erectile dysfunction were accepted and di
会议
Effect of dicycloplatin,a novel platinum chemotherapeutical drug,on inhibiting cell growth and induc
Dicycloplatin,a new supramolecular platinum antitumor drug which was formed by one molecule carboplatin and one molecule 1,1-ring succinic acid via the effect of hydrogen bond.In this study,we investi
会议
apoptosisdicycloplatincarboplatintumorcell growth
Enhancing Chemosensitivity in ABCB1-and ABCG2-Overexpressing Cells and Cancer Stem-Like Cells by An
Imidazopyridine CCT129202 is an inhibitor of Aurora kinase activity and displays favorable antineoplastic effect in preclinical studies.Here,we investigated the enhanced effect of CCT129202 on the cyt
会议
CCT129202Multidrug resistanceATP-binding cassette transportersAurora kinase i
Cyclin-dependent kinase 7/9 inhibitor SNS-032 abrogates FIP1-like-1 platelet-derived growth factor α
Purpose:The "gate-keeper" mutations T6741 PDGFRα in hypereosinophilic syndrome (HES) and T3151 Bcr-Abl in chronic myeloid leukemia (CML),are resistant to imatinib and the second-generation small molec
会议
SAHA and PJ 1548,a Novel Tyrosine Kinase Inhibitor Synergistically Induce Apoptosis in Imatinib-Resi
Limited treatment options are available for chronic myeloid leukemia (CML) patients who develop imatinib mesylate resistance particularly in patients with gatekeep mutant Bcr-Abl such as T3151.Compoun
会议
Curcumin and its Derivatives Potently Inhibits the T3151 Mutant and Overcomes Mutation-Based Resista
Inhibition of BCR-ABL by imatinib induces durable responses in many patients with chronic myeloid leukemia (CML),but resistance attributable to kinase domain mutations can lead to relapse.Mutation of
会议
Hsp90 N-terminal inhibitor 17-AAG and C-terminal inhibitor cisplatin synergistically eradicates Bcr-
Hsp90 is a ubiquitous molecular chaperone that regulates over 200 client proteins involved in multiple growth and signaling pathways,which are targets for chemotherapeutic strategies.Thus,Hsp90s role
会议
MTA3 and the Mi-2/NuRD complex regulates EMT in esophageal squamous cell carcinoma through chromatin
Epithelial-mesenchymal transition (EMT) is a key regulator for morphogenesis during embryonic development and the essential contribution of the EMT to carcinoma progression is the loss of their epithe
会议