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中科院上海生科院营养所陈雁研究组在一项研究中,阐明了糖原代谢关键蛋白PPP1R3G在维持葡萄糖稳态以及肝脏脂肪代谢中的作用。近日,相关研究论文在线发表于《分子内分泌学》。据介绍,机体利用多种机制参与餐后血糖调控,其中最主要的途径是通过胰岛细胞分泌胰岛素,增加机体外周组织对血糖的吸收,从而降低血糖。实验表明,进食后约三分之一的血糖能够转化为肝糖原。但目前尚不清楚肝脏的糖原合成如何与进食和饥饿的循环周期相协调,以维持餐
Chinese Academy of Sciences, Shanghai Institutes of Health Nutrition Chen Yan research group in a study elucidated the glycogen metabolism key protein PPP1R3G in maintaining glucose homeostasis and liver fat metabolism in the role. Recently, related research papers published online in “Molecular Endocrinology.” According to reports, the body uses a variety of mechanisms to participate in postprandial blood glucose regulation, the most important way is to secrete insulin through islet cells, increase the body’s peripheral tissues on the absorption of blood glucose, thereby reducing blood sugar. Experiments show that about one-third of the blood glucose after eating can be converted to glycogen. However, it is unclear how the liver’s glycogen synthesis is coordinated with the cycle of eating and starving to maintain meals