采用失血性休克(MAP6.0 kPa 90 min)回输失血复苏模型,在休克30 min 后治疗 Ver 处理组静脉漓注维拉帕米10 μg/kg·min)×15 min。休克组观察休克复苏4 h 后心、肺、肝、肾、胰、小肠等重要脏器组织超微结构及组织中黄嘌呤氧化酶和丙二醛含量的变化。透射电镜观察显示,休克对照组所有被观察的器官组织超微结构均呈严重的损伤性变化,其共同特点是线粒体减少及程度不同的损伤性改变,出现线粒体肿胀,基质中出现 Hemorrhagic shock (MAP6.0 kPa 90 min) was used to transfuse the hemorrhagic resuscitation model and verapamil (10 μg / kg · min) for 15 min after the treatment for 30 min in shock group. The shock group was observed 4 h after shock resuscitation heart, lung, liver, kidney, pancreas, small intestine and other important organs and tissue ultrastructure and tissue xanthine oxidase and malondialdehyde content changes. Transmission electron microscopy showed that all ultrastructures of organ tissues in the shock control group showed serious damage changes. Their common features were mitochondrial reduction and different degree of damage changes, mitochondria swelling and matrix appearance