迟发性皮肤卟啉病具有某些皮肤和生物化学特征.于暴露部位可出现水疱、粟丘疹、瘢痕、色素沉着、多毛及硬皮病样斑块.尿液常见尿卟啉过多,一般为两型:一型表现为肝(有时还有红细胞)缺乏尿卟啉原脱羧酶为特征者,其遗传素质可因酒精中毒、雌激素或肝病诱发,另一型在接触六氯苯或者多氯酚类后发生,但无遗传性酶缺乏.本文病例 Delayed skin porphyria with certain skin and biochemical characteristics in the exposed parts may appear blisters, milia, scars, pigmentation, hirsutism and scleroderma-like plaque. Urine urine uroporphyrin common too Two types: one type is characterized by liver (and sometimes erythrocytes) lack of uroporphyrinogen decarboxylase characterized by genetic predisposition may be due to alcoholism, estrogen or liver disease induced, the other type of exposure to hexachlorobenzene or more After the occurrence of chlorphenols, but no genetic enzyme deficiency in this case