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阻塞性肺疾病(OPD),尤其是哮喘病,死亡率和发病率非常高。自从了解了自身稳态机制的生理学以来,发病率和死亡率开始下降。对有关控制机制的了解,大大促进了对副作用小而有效的支气管扩张剂的研究。支气管和细支气管壁内的平滑肌、受自主神经系统支配,通常在自主神经系统(ANS)副交感神经通过迷走神经发放冲动导致支气管收缩,而交感神经通过β_2肾上腺能受体发放冲动引起支气管扩张。另外,还有一种非肾上腺能的神经支配也能引起支气管扩张。这一系统既不是交感神经也不是副交感神经系统,它可
Obstructive pulmonary disease (OPD), especially asthma, has a very high mortality and morbidity. Since understanding the physiology of its homeostasis mechanism, morbidity and mortality have started to decline. Understanding of the relevant control mechanisms has greatly facilitated the study of small and effective bronchodilators with fewer side effects. Bronchial and bronchial smooth muscle within the bronchiole wall, at the disposal of the autonomic nervous system, usually causes bronchoconstriction in the parasympathetic nerve of the autonomic nervous system (ANS) through vagus nerve firing, and the sympathetic nerves release bronchodilation through the impulse of beta 2 adrenergic receptors. In addition, there is also a non-adrenergic innervation can also cause bronchiectasis. This system is neither sympathetic nor parasympathetic nervous system, it can