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氩激光小梁成形术对小梁网的确切作用,尚未完全明了。业经大量资料证实,其降压效果系通过改善房水外流。从临床上看,该效应似与激光的特性(包括:波形、波长、烧灼部位、治疗时间,甚至在一定程度上还包括激光的能量)均不具特异性。其降压反应延迟发生,随时间减弱,有些病例可因再次治疗而重新获得降压效果。激光光凝对小梁网在聚焦焦点上产生破坏,但漫射则可兴奋小梁网细胞。激光治疗后小梁网形状改变,只有在眼压很高时才可能有助于房水外流。很可能细胞兴奋激活某种分子生物链变化,或许在小梁细胞外间质,从而改善房水外流易度。
The exact role of argon laser trabeculation in trabecular meshwork has not been fully understood. After a large number of data confirmed that the antihypertensive effect is through the improvement of aqueous humor outflow. Clinically, this effect appears to be nonspecific with the characteristics of the laser (including the waveform, wavelength, burning site, treatment time, and even to a certain extent, the energy of the laser). The antihypertensive response delay occurs, with the weakened in some cases due to re-treatment and regain the antihypertensive effect. Laser photocoagulation traverses the trabecular meshwork at the focal point of focus, but diffusion can excite the trabecular meshwork cells. Laser treatment of trabecular meshwork changes, only when the intraocular pressure is high may be helpful to the outflow of aqueous humor. It is possible that the cell is excited to activate some sort of molecular biological chain, perhaps in the extracellular matrix of the trabecular meshwork, thereby improving the atrial outflow ease.