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刺激家兔迷走神经1小时,肺磷脂含量增加可达31％。刺激猫的星状神经节,会引起肺静态顺应性变化。这些现象表明植物性神经对肺表面活性物质有调控作用。鉴于神经末梢与肺Ⅱ型上皮细胞之间的连接或接触一直未能在形态学上得到证实,Decamara等提出其调控机制可能有植物性神经-激素-肺Ⅱ型细胞或植物性神经-神经介质-肺Ⅱ型细胞二种方式。激素调控方式已有文介绍,现就神经介质调控方式作一简述。一、肾上腺素能介质给孕羊静脉滴注盐酸肾上腺索(3.3μg/min),6小时后检测到胎羊呼吸道液体中肺表面活性物质的含量增加。Abdellatif等给两组出生1-3天的家兔
Stimulate rabbits vagus nerve 1 hour, lung phospholipid content increased up to 31%. Stimulation of the cat’s stellate ganglion causes static changes in lung compliance. These phenomena suggest that vegetative nerves have a regulatory effect on pulmonary surfactant. In view of the fact that the connection or contact between nerve endings and type II epithelial cells has not been confirmed morphologically, Decamara et al proposed that its regulatory mechanism may include vegetative neuro-hormone-pulmonary type II cells or autonomic nerve-nerve mediators Lung Ⅱ type cells in two ways. Hormonal control methods have been introduced, now a brief description of the neural media control methods. First, the adrenergic mediator intravenous infusion of adrenal cord hydrochloride (3.3μg / min) to pregnant sheep, 6 hours after the detection of fetal sheep airway fluid lung surfactant content increased. Abdellatif et al gave two groups of rabbits born 1-3 days