尿毒症和接受肾移植的病人均可有甲状旁腺机能亢进(下称甲旁亢),其治疗以手术为主。近年来对此已有不少研究及报道,现作一综述。尿毒症病人继发性甲旁亢发生率和发病杌理用低铝透析液作长期血液透析(血透)的病人中,根据骨活检发现继发性甲旁亢的发生率高达67％,骨软化为23％。由于低血钙长期刺激甲状旁腺,甲状旁腺激素(PTH)的分泌增加,从而引起旁腺增生。此外,血清PTH增高的另一原因可能是肾衰竭末期的肾和肝对PTH的分解代谢能力降低。尿毒症中发生低血钙的机理则是血磷酸盐过多和继 Uremia and renal transplant patients may have hyperparathyroidism (hereinafter referred to as hyperparathyroidism), the treatment of surgery-based. There have been quite a few studies and reports on this in recent years. In patients with uremia, the incidence of secondary hyperparathyroidism and morbidity management with low-aluminum dialysis fluid for long-term hemodialysis (hemodialysis) patients, according to the biopsy found that the incidence of secondary hyperparathyroidism as high as 67%, bone Softened to 23%. As hypocalcemia long-term stimulation of parathyroid, parathyroid hormone (PTH) secretion increased, causing parathyroid hyperplasia. In addition, another cause of elevated serum PTH may be the renal and liver decomposing ability of PTH at the end of renal failure. The mechanism of hypocalcemia in uremia is hyperphosphatemia and follow-up