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本文用神经电生理方法研究了氰戊菊酯、氯菊酯对棉铃虫Helicoverpaarmigera（Hubner）相对敏感（HD－S）种群和抗性（HJ－R）种群的神经毒理作用。10－5mol／L的氰戊菊酯、10－5mol／L，的氯菊酯诱发腹神经索自发发放频率的增加和随后的神经传导阻断，10-4mol／L的氯菊酯抑制HD－S种群的神经兴奋，直接阻断神经传导。以兴奋时间、神经传导阻断时间、对药剂作用反应时间的个体分布频率3个参数比较两种群对杀虫剂的反应，均发现HJ－R种群比相对HD-S种群表现了2～3倍的神经不敏感机制，并且发现这种神经不敏感机制对毒理Ⅰ型和Ⅱ型拟除虫菊酯同样有作用。
In this paper, the neurotoxicity of fenvalerate and permethrin against the HD-S and HJ-R populations of Helicoverpa armigera (Hubner) was studied by electrophysiological methods. 10-5mol / L fenvalerate, 10-5mol / L, permethrin induced spontaneous release of abdominal nerve cable increased frequency and subsequent blockade of nerve conduction, 10-4mol / L permethrin HD- S-neuronal excitations directly block nerve conduction. The results showed that HJ-R population was 2-3 times more sensitive than HD-S population in terms of excitement time, nerve conduction block time and frequency of individual distribution of response time of the drug. Insensitive mechanism of neurotoxicity and found that this neuronal insensitive mechanism has the same effect on toxicological types I and II pyrethroids.