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总结分析７２例急性水肿型胰腺炎合并胃损害的情况。结果显示６２５％（４５／７２）的患者伴有活动性胃炎和消化性溃疡，前者的发生率为４８６％，后者为１３８％。胆源性和酒精源性胰腺炎合并胃损害的发生率分别为６８３％和６０７％，无显著性差异。轻度胰腺炎合并胃损害的发生率低于中、重度的发生率。血清胃泌素无明显变化，幽门螺杆菌（Ｈｐ）的感染率为３７８％。结果表明急性水肿型胰腺炎合并胃损害的发生可能主要与胰蛋白酶、溶血磷脂活性增加，使胃粘膜血流减少，从而导致胃粘膜抗力下降等因素有关，而与胃泌素、酒精刺激及Ｈｐ的感染关系不大。此外，合并的胃损害可能是急性水肿型胰腺炎腹痛消失较慢的原因。
Summary of 72 cases of acute edematous pancreatitis associated with gastric damage. The results showed that 62.5% (45/72) of patients with active gastritis and peptic ulcer, the former was 48.6%, the latter was 13.8%. The incidence of biliary and alcohol-induced pancreatitis with gastric lesions were 683% and 607%, respectively, with no significant difference. The incidence of mild pancreatitis with gastric damage is lower than the incidence of moderate and severe. No significant changes in serum gastrin, Helicobacter pylori (Hp) infection rate was 37 8%. The results showed that acute edematous pancreatitis with gastric damage may occur mainly with trypsin, lysophospholipid activity increased gastric mucosal blood flow decreased, leading to decreased resistance to gastric mucosal factors and other factors, and gastrin, alcohol stimulation and Hp The infection is not related. In addition, the combined gastric damage may be the cause of the slower disappearance of abdominal pain in acute edematous pancreatitis.