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张口受限可能由于下颌喙突的伸长而与相应的颧骨发生机械性锁结,至今仍认为这种情况少见。但作者在1987年报道过在一组张口受限病人的纵向观察中,喙突锁结的发生率达5%。喙突的伸长是TMJ内部结构紊乱造成的下颌动度降低后喙突上颞肌牵拉反应性骨质增生联合作用的结果,本文的目的是揭示病人喙突增大及锁结与颅面形态大小的关系。材料和方法本研究包括19名因喙突增大和颧骨发生锁结而造成张口受限的患者,张口度从1～29mm,X线断层摄影证实为骨与骨锁结,其中3例因有左右两边广泛性的不对称颌骨颅面异常而去除。剩余16名分为先天性组及诱发组。
Limited mouth opening may be due to the elongation of the mandibular coracoma and the corresponding zygomatic mechanical lock occurs, still considered this rare. However, in 1987, the authors reported that the incidence of coracoid locking was 5% in a longitudinal view of a group of restricted mouths. The coracoid process is the result of combined action of TMJ-induced temporal hyperosmolar hyperosteogeny induced by mandibular mobility caused by disorder of internal structure of TMJ. The purpose of this paper is to reveal the increase of coracoid process, The size of the relationship. Materials and Methods The study included 19 patients with limited mouth opening due to the increased coracoid process and the zygomatic junction. The mouth opening ranged from 1 to 29 mm. X-ray tomography confirmed bone-to-bone interlocking. Three Left and right extensive asymmetric craniofacial abnormalities removed. The remaining 16 were divided into congenital group and induced group.