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病毒性心肌炎(viral myocarditis,VMC)为儿科临床常见的后天获得性心脏病,其发病机制包括:病毒的直接损伤、免疫机制、一氧化氮(nitric oxide,NO)的作用以及能量代谢机制。柯萨奇B组病毒(coxsackie virus,CVB)为其主要的病原体[1]。同时腺病毒在VMC患者中也有很高的检出率[2]。现就VMC发病机制的研究进展综述如下。
Viral myocarditis (VMC) is a common clinical acquired heart disease in pediatrics. Its pathogenesis includes direct virus damage, immune mechanism, the role of nitric oxide (NO) and energy metabolism. Coxsackie virus (coxsackie virus, CVB) as its main pathogen [1]. At the same time adenovirus in VMC patients also have a high detection rate [2]. Now on the pathogenesis of VMC research progress are summarized below.