Extracellular Signal-Regulated Kinase in Nucleus Accumbens Mediates Propofol Self-Administration in

来源 :神经科学通报(英文版) | 被引量 : 0次 | 上传用户:kooksnake
下载到本地 , 更方便阅读
声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架
论文部分内容阅读
其他文献
Dear Editor,rnComa,the vegetative state (VS),and the minimally-conscious state (MCS),often collectively referred to as disorders of consciousness (DOCs),typically occur after severe traumatic or non-traumatic brain injury [1].The boundary between awarenes
期刊
Dear Editor,rnRecently,several single nucleotide polymorphisms (SNPs;rs34094401 on RAD51B,rs41309351 on CPXM1,rs143555311 on MPHOSPH10,rs141620200 on SER-PINA1,and rs2424913 on DNMT3B) have been associated with Parkinson\'s disease (PD) in Caucasians [1
期刊
Alzheimer\'s disease (AD),the most common type of dementia,has been identified as a protein misfolding disease with the accumulation of abnormally folded amyloid-β (Aβ) protein and hyperphosphorylated tau protein in the brain [1,2].Aβ is known to play a
期刊
Neurons,especially axons,are metabolically demanding and energetically vulnerable during injury.However,the exact energy budget alterations that occur early after axon injury and the effects of these changes on neuronal survival remain unknown.Using a cla
SOCS3,a feedback inhibitor of the JAK/STAT signal pathway,negatively regulates axonal regrowth and inflammation in the central nervous system (CNS).Here,we demonstrated a distinct role of SOCS3 in the injured spinal cord of the gecko following tail amputa