论文部分内容阅读
中枢神经系统感染性疾病发生后,由于发热、昏迷、脑水肿,使组织缺氧、酸中毒、氧自由基产生,细胞内钙离子积聚以及颅内血管的炎性改变等均影响了正常的血液流变学,结果损害脑血流及微循环灌注,造成脑组织缺血缺氧,加速脑水肿的进展。本文检测24例中枢神经系统感染性疾病的红细胞压积、红细胞沉降率、红细胞沉降率方程K值、血浆纤维蛋白原、红细胞电泳时间、血小板电泳时间、血浆粘度和切变率分别为4s~(-1)、10s~(-1)、20s~(-1)、40s~(-1)、100s~(-1)、200s~(-1)时的全血表现粘度值,就其与中枢神经系统感染性疾病的关系作一初步的探讨与分析。
Central nervous system infectious diseases, due to fever, coma, cerebral edema, hypoxia, acidosis, oxygen free radicals, intracellular calcium accumulation and intracranial vascular inflammatory changes affect the normal blood Rheology, the results of damage to cerebral blood flow and microcirculation perfusion, resulting in ischemia and hypoxia of brain tissue, accelerate the progress of cerebral edema. In this paper, 24 cases of central nervous system infectious disease hematocrit, erythrocyte sedimentation rate, erythrocyte sedimentation rate equation K value, plasma fibrinogen, erythrocyte electrophoresis time, platelet electrophoresis time, plasma viscosity and shear rate were 4s ~ ( (-1), 10s -1, 20s -1, 40s -1, 100s -1 and 200s -1, Neurological Infectious Diseases for a preliminary discussion and analysis.