兔第四脑室给予血管紧张素Ⅱ(2μg/20μl),引起兔子均动脉血压升高和心率减慢,后者不是血管紧张素Ⅱ本身的作用,而是压力感受器反射性影响的后果。热烫损毁后极区(area poctrema),血管紧张素Ⅱ的加压作用减弱,但未完全消失。提示后极区只参与部分作用。实验还证明,纳洛酮对血管紧张素Ⅱ的加压作用无影响,提示麻醉状态下内源性阿片肽和血管紧张素Ⅱ之间无关系。 Angiotensin II (2μg / 20μl) given to the fourth ventricle in rabbits caused a rise in mean arterial blood pressure and heart rate in rabbits. The latter was not the result of angiotensin II itself, but the consequence of baroreflex reflexes. In the area after psoriasis, the pressure of angiotensin Ⅱ was weakened, but it did not disappear completely. Polar region prompted only part of the role. Experiments also show that naloxone has no effect on the pressure of angiotensin Ⅱ, suggesting that there is no relationship between endogenous opioid peptide and angiotensin Ⅱ under anesthesia.