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实验动物给予镉后,在蓄积金属的器官可诱导金属硫蛋白的生成,特别是肝脏和肾脏。这种金属硫蛋白也可导致锌和铜在组织的潴留。因为镉的蓄积可引起锌和铜内环境稳定的明显改变,结果使得其他生化过程中金属的效能减低,并可能引起组织衰竭。已知慢性镉接触可使肝脏铁的贮存降低,以及血浆中金属含量的减少。在其他毒性方面,长期接触镉可产
After the experimental animals were given cadmium, metal-accumulating organs could induce the production of metallothionein, especially liver and kidney. This metallothionein can also lead to the retention of zinc and copper in the tissues. Because the accumulation of cadmium can lead to significant changes in the stability of zinc and copper within the environment, the result of other biochemical processes to reduce the effectiveness of metals, and may cause tissue failure. It is known that exposure to chronic cadmium reduces the storage of iron in the liver, as well as the reduction of metal content in the plasma. In other toxicities, prolonged exposure to cadmium can produce