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慢性肾功能衰竭尿毒症患者的感染发生率明显增高,提示其存在免疫功能低下,但慢性肾衰尿毒症患者免疫缺陷的机制仍不十分清楚。本文利用单克隆抗体(McAb)初步观察了慢性肾衰(CRF)大鼠T细胞亚群的变化,试图揭示慢性肾衰尿毒症免疫缺陷的机制,为临床治疗提供理论依据。
Chronic renal failure patients with uremia infection was significantly higher incidence, suggesting that there is immune dysfunction, but the mechanism of immune deficiency in patients with chronic renal failure uremia is still not very clear. In this paper, monoclonal antibody (McAb) was used to observe the changes of T cell subsets in chronic renal failure (CRF) rats in order to reveal the mechanism of immune deficiency in chronic renal failure and provide a theoretical basis for clinical treatment.