论文部分内容阅读
我院于77～79年间曾收治急性氰化物中毒继发尿崩症、急性苯胺及硝基苯中毒继发精神性多饮多尿症各一例。这种情况未见文献报告,故予以介绍。供同道参考。 例1:王××,男,28岁,住院号5119,操作工。于1977年9月18日因工作不慎吸入了高浓度氰化氢气体数口,随即意识丧失,昏倒在地。经吸氧、亚硝酸异戊酯吸入、静咏注射美兰及硫代硫酸钠35分钟后清醒。醒后全身颤抖,自述头晕、头痛、全身无力并恶心呕吐。体检:体温37.4℃,脉搏96次/分,呼吸20次/分,血压120/80mmHg、心肺正常。腹部平软,肝脾未触及。生理反对存在,未引出病理反射。尿常规正常。肝功及转氨酶正常。既往健康。无家族遗传性疾病。经解毒及对症治疗上述症状好转。
Our hospital in 77 to 79 years had received acute cyanide poisoning secondary to diabetes insipidus, acute aniline and nitrobenzene secondary mental polyhydramnios a case of each. This situation has not been reported in the literature, so be introduced. For fellow reference. Example 1: Wang XX, male, 28 years old, hospital number 5119, operator. On September 18, 1977 due to work inadvertently inhaled a few high-concentration hydrogen cyanide gas mouth, then loss of consciousness, collapsed to the ground. After oxygen, isoamyl nitrite inhalation, intravenous injection of melanin and sodium thiosulfate 35 minutes after awake. Whole body tremble after wake up, readme dizziness, headache, general weakness and nausea and vomiting. Physical examination: body temperature 37.4 ℃, pulse 96 beats / min, breathing 20 beats / min, blood pressure 120 / 80mmHg, normal heart and lung. Abdomen soft, liver and spleen not touched. Physiological opposition exists, did not lead to pathological reflex. Urine routine normal. Liver function and transaminase normal. Past health. No family genetic disease. After detoxification and symptomatic treatment of the above symptoms improved.