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青光眼性视神经萎缩由异常的眼内压所致。用白/黑(W/B)模型刺激,视诱发电位(VEP)潜伏期延长与杯盘比(C/D)相关。在C/D大于0.8眼,VEP三相波均有延迟。同样,异常的VEP振幅也发生在C/D大于0.8的眼。在这些病例中,VEP三相波振幅降低的程度与C/D的扩大相一致。用红/黑(R/B)模型刺激,比较患者的双眼VEP。在一眼C/D比另一眼大0.2的眼,VEP的潜伏期和振幅的异常比另一眼增加。我们研究表明,颜色模型VEP可很好地检测青光眼性视神经萎缩的功能。此技术对于研究VEP与由压力性水肿和其他因素引起的视神经异常之间的关系是有用的。
Glaucomatous optic atrophy is caused by abnormal intraocular pressure. Stimulation with a white / black (W / B) model resulted in an increase in the latency of the apparent evoked potential (VEP) associated with the cup-to-cup ratio (C / D). In C / D greater than 0.8 eye, VEP three-phase wave is delayed. Similarly, abnormal VEP amplitude also occurs in C / D greater than 0.8 eye. In these cases, the amplitude of the VEP three-phase wave was reduced to the same extent as the C / D expansion. Stimulated with a red / black (R / B) model comparing the patient’s binocular VEPs. At a glance of 0.2 in the C / D compared to the other eye, the latency and amplitude abnormalities of VEP increased more than the other eye. Our research shows that the color model VEP can detect the function of glaucomatous optic atrophy well. This technique is useful for studying the relationship between VEP and optic nerve abnormalities caused by pressure edema and other factors.