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近十年来,由于对淋巴细胞的深入研究,大大促进了免疫学的进展,同时对淋巴肿瘤本质的认识已进入了一个新阶段。自1973年Lukes报导了原始免疫细胞性淋巴结病(Immunoblastic Lymphadenop-athy简称IBL)和1974年Frizzera等报导了血管原始免疫细胞性淋巴结病伴发异常蛋白血症(Angioimmunoblastic Lym-phadenopathy With Dysproteinemia简称AILD)后,近来已将其作为一个新发现的独立的疾病,其基本过程为B细胞系统的过度免疫性增殖,由于某种抗原激发淋巴细胞过度转化为原始免疫细胞和浆细胞的过敏反应。
In the past ten years, due to in-depth study of lymphocytes, greatly promoted the progress of immunology, while the understanding of the nature of lymphoma has entered a new phase. Since 1973, Lukes reported that the primitive immunocytic lymphadenopathy (IBL) and 1974, Frizzera et al reported vascular primed immune cell lymphadenopathy with abnormal proteinuria (Angioimmunoblastic Lym-phadenopathy With Dysproteinemia referred to as AILD) Recently, it has been used as a newly discovered independent disease. The basic process is the over-immunity proliferation of B-cell system. Some antigens stimulate the over-conversion of lymphocytes into allergic reaction of primitive immune cells and plasma cells.