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由于耐淀粉酶淀粉在结肠中经细菌发酵后可转变为短链脂肪酸,并且在实验中证实粘膜环腺苷酸不会改变短链脂肪酸促进钠吸收,因而假设在治疗霍乱用的标准口服补盐溶液(ORS)中添加耐淀粉酶淀粉可能会减少粪便中液体的丢失量及缩短腹泻持续时间。作者的研究旨在证实这一假设的正确性。 研究对象为1994年5月～1996年7月期间序贯入院的48例霍乱患者(14～58岁)。所有患者的粪便中均检出霍乱弧菌;急性水样便腹泻持续时间小于72小时。入院时有低血压者静脉补液后列入研究,但曾用抗生素治疗或有严重全身性疾病的患者除外。
Since amylase-resistant starch can be transformed into short-chain fatty acids after bacterial fermentation in the colon and it has been experimentally demonstrated that mucosal cyclic adenosine does not alter short-chain fatty acids for sodium absorption, it is hypothesized that the standard oral rehydration salts used in the treatment of cholera Adding amylase-resistant starch to the solution (ORS) may reduce the amount of liquid lost in the stool and shorten the duration of the diarrhea. The author’s study aims to confirm the validity of this hypothesis. The subjects were 48 cholera patients (14-58 years) with sequential admission from May 1994 to July 1996. Vibrio cholera was detected in all patients’ faeces; acute watery diarrhea lasts for less than 72 hours. Hypotension was admitted to hospital after intravenous rehydration studies, but once treated with antibiotics or patients with severe systemic disease except.