锰矿工人尘肺在文献上报导较少,国内仅1965年鞍钢劳研所有过报导。一般认为系锰尘和二氧化矽粉尘引起的混合尘肺,发病率较低,为慢性进展的纤维化改变。其表现与一般尘肺相同,主要为肺间质的纤维增生,肉芽肿变化。肺泡间隙增厚,支气管及血管周围不规则的纤维化。其 X 线特点根据苏联作者报导为细小病灶与网状改变相结合,并有肺门增大征。 Manganese miners pneumoconiosis reported in the literature less, only 1965 Anshan Iron and Steel Research Institute have reported. It is generally believed that mixed dust pneumoconiosis caused by manganese dust and silica dust has a lower incidence and is a fibrosis change which is chronic progression. The performance of the same with the general pneumoconiosis, mainly interstitial lung fibrosis, granuloma change. Alveolar interstitial thickening, bronchial and perivascular irregular fibrosis. The X-ray features are based on a combination of small lesions and reticular changes reported by the Soviet Union authors and have hilar augmentation.