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实验选用自然老龄大鼠和 O_3损伤小鼠作为衰老模型,经用“补血Ⅰ号”药物治疗后,结果大鼠脑、肾组织和小鼠肝组织过氧化物含量明显降低;同时能使小鼠肝组织超氧化物歧化酶的活性明显增高。
In the experiment, natural aged rats and O_3 injured mice were used as an aging model. After treatment with “Blood No. I”, the peroxide content in rat brain, kidney tissue, and mouse liver tissues was significantly reduced; Superoxide dismutase activity in liver tissue was significantly increased.