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给予磺柳硝胺后，离体兔胸主动脉血管环标本呈剂量依赖性舒张，此作用被亚甲蓝阻断而与血管内皮存在与否无关。结果显示，磺柳硝胺释放一氧化氮发挥舒血管作用。
Given sulfoneamide, isolated rabbit thoracic aortic vascular specimens in a dose-dependent manner, this role is blocked by methylene blue and vascular endothelial existence or not. The results showed that sulfonamide released nitric oxide to exert vasodilatation.