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患者男,30岁。于1990年9月24日15时因家中煤气软管脱落致神志不清,立即抬入我院急诊。查体:深昏迷状态,压眶上切迹无反应,瞳孔等大圆,对光反应迟钝,角膜反射迟钝。左巴彬斯基征阳性。化验室检查:血WBC14.8×10~9/L,血BUN26mg％,EKG提示不完全右束支传导阻滞。入院后第15天,CT揭示“脑白质密度降低符合一氧化碳中毒改变”,诊断;急性重度一氧化碳中毒。治疗以改善脑微循环,增强脑细胞代谢,清除自由基,高压氧治疗为重点。低右500ml加入A.T.P.40mg,CoA100~u,Cytc30mg,VitB_6100mg,VitB_(12)250mg,或
Male patient, 30 years old. At 15:00 on September 24, 1990 due to loss of gas hose at home caused by confusion, immediately carried into our hospital emergency room. Physical examination: deep coma state, no effect on the supraorbital notch, pupils and other great circles, slow response to light, corneal reflex slow. Left Babinski sign positive. Laboratory tests: blood WBC14.8 × 10 ~ 9 / L, blood BUN26mg%, EKG prompted incomplete right bundle branch block. On the 15th day after admission, CT revealed that “the reduction of white matter density conforms to the change of carbon monoxide poisoning”, diagnosis; acute severe carbon monoxide poisoning. Treatment to improve cerebral microcirculation, enhance brain cell metabolism, free radical scavenging, hyperbaric oxygen therapy as the focus. Low right 500ml A.T.P. 40mg, CoA100 ~ u, Cytc30mg, VitB_6100mg, VitB_ (12) 250mg, or