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目的和方法：给大鼠造成３０％体表面积的全层烧伤并延迟输液复制大鼠烧伤后高代谢伴多器官功能不全的模型，观察该模型动物血浆一氧化氮（ＮＯ）及枯否细胞产生ＮＯ的量。结果：烧伤后大鼠血浆ＮＯ含量升高，并与其中ＴＮＦα的含量呈正相关。烧伤后大鼠枯否细胞在受ＬＰＳ刺激下ＮＯ的含量明显高于对照组。结论：ＮＯ在烧伤后器官功能损伤中具有一定作用
Aims and Methods: A total burn of 30% of the body surface area was induced in rats, and the model of hypermetabolism with multiple organ dysfunction after infusion of the infusion solution was delayed. The levels of plasma nitric oxide (NO) and Kupffer cells The amount of NO. Results: The content of NO in plasma increased after burn, and it was positively correlated with the level of TNFα. The content of NO in Kupffer cells stimulated by LPS was significantly higher than that in control group. Conclusion: NO plays a role in organ dysfunction after burn injury