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肺癌是人类最常见的恶性肿瘤之一,其发生、发展和转移是一个极其复杂的多基因调控异常的过程[1]。研究表明,肺癌的发生是由于某些原癌基因的激活、抑癌基因的失活和凋亡相关基因的改变导致细胞增殖和死亡异常的结果[2]。基因的异常先于形态学改变,因此检测发生率高,特异性强的
Lung cancer is one of the most common malignant tumors in humans. The occurrence, development and metastasis of lung cancer are an extremely complicated process of multi-gene regulation abnormalities [1]. Studies have shown that the occurrence of lung cancer is due to the activation of certain oncogenes, inactivation of tumor suppressor genes and apoptosis-related gene mutations lead to abnormal cell proliferation and death results [2]. Gene abnormalities precede the morphological changes, so the detection of high incidence and specificity