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本实验采用神经元单位放电记录的方法，在Ｗｉｓｔａｒ大鼠上观察到网状巨细胞核伤害反应性神经元４２个，其中伤害兴奋性神经元（ＮＥＮ）３４个，伤害抑制性神经元（ＮＩＮ）８个。电刺激黑质对多数ＮＥＮ和ＮＩＮ放电呈抑制作用，其值分别由刺激黑质第ｌｍｉｎ时的１８．８±４．４和１７．５±３．５，下降到第５ｍｉｎ时的１．９±１．３和４．１±２．３（Ｐ＜０．０１），反应呈递减下降。刺激黑质对少数ＮＥＮ和ＮＩＮ的放电有兴奋作用，其值分别由电刺激黑质第１ｍｉｎ时的１２．４±３．２和１７．８±４．８，升高到第５ｍｉｎ时的１８．５±４．１和２５．５±２．０时（Ｐ＜０．０１）．氟哌啶醇注入单侧ＰＡＧ腹外侧部或电解损毁同一部位可以阻断刺激黑质的效应。这提示黑质对网状巨细胞核的影响是通过ＰＡＣ发挥作用的，而且ＳＮ－ＰＡＧ末梢的递质是多巴胺能。
In this study, 42 neurons of reticular giant cell nuclear injury were observed in Wistar rats, including 34 neurons injured by excitatory neurons (NEN), injury-inhibiting neurons (NIN) 8 Stimulation of substantia nigra for most of the NEN and NIN discharge was inhibited, the value of the first lmin to stimulate the substantia nigra at 18.8 ± 4.4 and 17.5 ± 3.5, down to 5min at 1.9 ± 1.3 and 4.1 ± 2.3 (P <0.01), the response decreased and decreased. Stimulation of substantia nigra stimulated the discharge of a few NEN and NIN, which were increased from 12.4 ± 3.2 and 17.8 ± 4.8 at 1 min of electrical stimulation of substantia nigra to 18 .5 ± 4.1 and 25.5 ± 2.0 (P <0.01). Haloperidol into the unilateral lateral ventricular PAG or electrolytic damage to the same site can block the stimulation of substantia nigra effects. This suggests that the effects of substantia nigra on the giant cytoplasmic reticulum play a role through the PAC and that the transmitter of SN-PAGs is dopaminergic.