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急性心肌梗塞(AMI)后心脏猝死(SCD)至今仍是一大难题。AMI急性期和亚急性期存活的患者在出院后第一年内大约有10%死亡,多数病例为SCD。关于SCD的机制多数学者认为是心律失常,在很大程度上室性心律失常意味着是患者临终前表现。然而,有学者提出,究竟室性心律失常是AMI患者本身的原发心电异常还是其他机制触发致命性心律失常的继发表现?自从对AMI后无症状患者使用Ⅰ类抗心律失常药抑制室性心律失常获阴性结果以来,这个问题才引起重视,早期认为这类患者是AMI后SCD的高危人群。
Sudden cardiac death (SCD) after acute myocardial infarction (AMI) is still a major challenge. In the acute and subacute AMI patients, approximately 10% died in the first year after discharge, with most cases being SCD. The mechanism of SCD Most scholars believe that arrhythmia, to a large extent, ventricular arrhythmia means that the patient’s performance before the last. However, some scholars have raised the question whether ventricular arrhythmias are the primary ECG abnormalities in patients with AMI or the secondary manifestation of other mechanisms triggering life-threatening arrhythmias. Since class I anti-arrhythmic agents are used asymptomatically in AMI patients, This problem has attracted much attention since the negative outcome of arrhythmia, which was previously considered a high-risk group of post-AMI SCD.