目前认为迟发型超敏反应(DTH)是曼氏血吸虫肺期免疫力的效应机制,由致敏T淋巴细胞释出的γ-干扰素激活巨噬细胞是DTH的一个内在特征,所以证实肺内激活的巨噬细胞将会增强DTH作为肺部效应机制这一事实。为此,作者使用20Kr照射致弱的500条曼氏血吸虫尾蚴接种C57BL/6小鼠(试验组),同时设置了用80Kr照射致弱的相同数量尾蚴接种组及未接种组小鼠作为对照。5周后三组小鼠均以,200条正常尾蚴 It is thought that delayed-type hypersensitivity (DTH) is the effector mechanism of Schistosoma mansoni’s lung immunity, and γ-interferon-activated macrophages released from sensitized T lymphocytes is an intrinsic feature of DTH, Activated macrophages will enhance the fact that DTH acts as a mechanism of lung effects. To this end, the author used 500kw of Schistosoma mansoni to inoculate C57BL / 6 mice (experimental group) with weak irradiation of 20Kr, and set the same number of cercariae inoculated group and unvaccinated group mice as controls with 80Kr irradiation weakened. After 5 weeks, the three groups of mice all had 200 normal cercariae