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为了给“抑制死”提供实验根据和理论依据，模拟“抑制死”的体表刺激，用3.0kg触压麻醉开胸犬的心前区、剑突下，同步记录大右心房神经节丛神经元内单位放电的频数及心脏变时。变力性反应。静脉给入普来洛尔（1mg／kg）或阿托品（1mg／kg）及心脏急性会神经支配后，重复上述刺激。结果显示：触压犬心前区可直接引起心脏表面神经节丛内的副交感神经元兴奋，窦房结自律性降低，引起负性频率作用；在此基础上刺激兴奋心脏表面交感神经元可诱发室性心律失常。提示刺激心前区、剑突下引起的“抑制死”，不仅与迷走神经对心脏的抑制有关，可能还与交感神经元对心室的兴奋有关。
In order to provide the experimental basis and theoretical basis for “inhibiting death”, simulated “inhibition of death” surface stimulation, with 3.0kg pressure anesthesia in the anterior pituitary of the thoracodorsal canine, simultaneous recording of the right atrial ganglion plexus Unit discharge frequency and heart changes. Variable reaction. The above stimulation was repeated after intravenous administration of either buprenolol (1 mg / kg) or atropine (1 mg / kg) and acute innervation of the heart. The results showed that the frontal area of dogs could directly stimulate the excitability of parasympathetic neurons in the ganglion plexus on the surface of the heart and reduce the self-regulation of the sinus node, resulting in the negative frequency. On the basis of this, stimulating excitable cardiac surface sympathetic neurons could be induced Ventricular arrhythmia. Prompted to stimulate the precordial, xiphoid caused by “inhibition of death”, not only with the vagus nerve inhibition of the heart, may also be sympathetic to the excitement of the ventricle.