论文部分内容阅读
目的 探讨肺癌致上腔静脉综合征(SVCS)生物学行为、肺癌与上腔静脉梗阻段周围病变位置关系的CT特征及病理学基础。资料与方法 病理确诊肺癌致SVCS共5 1例,回顾分析肺癌解剖部位、大体类型及其致SVCS生物学行为、与上腔静脉梗阻段周围病变间位置关系的CT表现,并作统计分析。结果 肺癌致SVCS生物学行为CT表现:直接蔓延7例,淋巴结转移17例,二者并存2 7例。致SVCS肺癌与上腔静脉梗阻段周围病变间位置关系CT表现:直接相连32例,间接相连10例,不相连接9例。不同肺叶不同大体类型肺癌致SVCS生物学行为及其与上腔静脉梗阻段周围病变间位置关系均不同(P <0 .0 5 )。结论 肺癌致SVCS生物学行为、与上腔静脉梗阻段周围病变间位置关系均与肺癌解剖部位及大体类型有关。
Objective To investigate the CT features and pathological basis of the biological behavior of lung cancer caused by superior vena cava syndrome (SVCS) and the relationship between lung cancer and the lesions around the superior vena cava obstruction. MATERIALS AND METHODS: Fifty-one cases of SVCS caused by lung cancer were retrospectively analyzed. The CT findings of the anatomical sites, general types, SVCS-induced biological behaviors and the location of the lesions around the superior vena cava obstruction were retrospectively analyzed and statistically analyzed. Results The biological behavior of SVCS induced by lung cancer showed 7 cases of direct spread and 17 cases of lymph node metastasis. The two groups coexisted in 27 cases. To SVCS lung cancer and superior vena cava lesions around the location of the relationship between lesions CT: directly connected to 32 cases, indirectly connected to 10 cases, not connected to 9 cases. The biological behaviors of different general types of lung cancer caused by different lung lobes and their relationship with the lesions around the superior vena cava obstruction segment were all different (P <0.05). Conclusions The biological behavior of SVCS induced by lung cancer and the location of the lesions around the obstruction of the superior vena cava are related to the anatomic site and the general type of lung cancer.