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测定了大鼠游泳60min和180min后血清、肌肉和肝组织亚细胞组分中脂质过氧化反应产物丙二醛的含量.结果发现,游泳60min后,大鼠血清、肌肉和肝细胞线粒体和微粒体中的丙二醛含量显著高于安静对照组和实验对照组,肌肉和肝细胞胞浆中的丙二醛含量在运动前后无显著性差异.游泳至180min时,上述组分中的丙二醛含量均进一步增加.表明一定时间的游泳运动使脂质过氧化代谢产物增多,这可能是导致运动能力下降、运动性损伤和疲劳的原因之一.
The content of malondialdehyde (MDA) in the subcellular fractions of serum, muscle and liver tissue after 60min and 180min swimming in rats was measured. The results showed that the contents of malondialdehyde in mitochondria and microsome of rat serum, muscle and liver cells were significantly higher than those of the control group and the control group after 60-minute swimming. The contents of malondialdehyde in the cytoplasm of muscle and hepatocyte were significantly decreased No significant difference before and after. The swimming time to 180min, the above components in the MDA content were further increased. This suggests that increased swimming of lipid peroxidation metabolites over time may be one of the causes of decreased exercise capacity, motor injury and fatigue.