本文通过对50例病态窦房结综合征(SSS)的临床分析,得出如下意见:一.窦房结病变是一种长期的慢性进行性的退行性改变,病程漫长,早期可仅表现为窦性心动过缓,而且窦缓可为间歇性,无特异性。SSS 进一步发展,窦房结不能产生冲动时可导致窦性静止或窦房阻滞,在极严重病例可完全或几乎完全没有窦性激动,从而导致心房停顿,代之以房室连接处性或室性逸搏心律。当病变累及心房或由于心动过缓影响心房的电生理性能时,可伴发阵发性房性心动过速、心房朴动和心房颤动。窦房结病变伴房室交接区病变称为“双结病变”,若兼有心房、交接区、房室束,束支及浦顷野氏纤维等部位的病变则称为“全传导系统缺陷”。本组患者有双结病变者7例,全传导系统病变2例。其中 This article through the clinical analysis of 50 cases of sick sinus syndrome (SSS), the following observations: I. Sinus lesions is a long-term chronic progressive degenerative changes, long course of disease, the early can only be manifested as Sinus bradycardia, and slow sinus can be intermittent, non-specific. SSS further development, sinus node can not produce impulses can lead to sinus arrest or sinoatrial block, in very serious cases can be completely or almost no sinus excitement, resulting in atrial pause, replaced by atrioventricular junctional sex or Room escape rhythm. When lesions affect the atrium or due to bradycardia affect the electrophysiological performance of the atria, may be associated with paroxysmal atrial tachycardia, atrial Park move and atrial fibrillation. Sinus node lesions with atrioventricular junction area lesions known as “double junction lesions”, if both atrial, junction area, atrioventricular bundle, bundle branch and Po are wild fibers and other parts of the lesion is called “full conduction system defects ”. This group of patients with double junction lesions in 7 cases, 2 cases of total conduction system lesions. among them