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目的：研究血流动力和肺淋巴引流对肺水的调节作用。方法：以扩容手段使山羊血流动力、肺淋巴引流和肺水发生改变，定时监测这些改变的参数并进行比较。结果：在各项血流动力参数中，外周血管阻力（SVR）的变化对肺水的影响最为显著。SVR下降不仅可以缓解体液向肺循环中转移，肺淋巴引流也有所增加。肺淋巴流出管全部结扎的动物，肺水肿的发生并不如预料之迅速，但SVR的下降却甚突出，可能即因此缓解了肺水的聚集。正常的血流动力虽是肺水的来源和运行动力，但其疏导肺水的作用却比较间接。肺淋巴液的疏导主要取决于肺血水重、总肺水量和血管外肺水的多寡，其他因素的影响并不显著。结论：肺水本身可能具有一定的（自身）调节能力。
Objective: To study the regulation of pulmonary water by hemodynamics and pulmonary lymphatic drainage. METHODS: Goat hemodynamics, pulmonary lymphatic drainage, and pulmonary water changes were scaled-up to monitor these changes regularly and compare them. Results: The changes of peripheral vascular resistance (SVR) had the most significant effect on the lung water in all hemodynamic parameters. SVR decline can not only ease the body fluid transfer to the pulmonary circulation, pulmonary lymphatic drainage has also increased. Pulmonary lymphatic outflow tube ligation of all animals, the occurrence of pulmonary edema is not as expected quickly, but the decline in SVR is even more prominent, which may therefore ease the pulmonary water aggregation. Although the normal blood flow of lung water is the source and operation of power, but its role in diverting lung water is more indirect. Pulmonary lymphatic drainage mainly depends on pulmonary blood flow, total lung volume and extravascular lung water, the other factors are not significant. Conclusion: Pulmonary fluid itself may have some (self) regulatory capacity.