论文部分内容阅读
作者报告2例成人急性脑梗塞,均患肾病综合征所致的高凝状态。例1男,36岁。因右侧肢体无力,不能讲话住院。检查有同向偏盲,右偏身感觉减退。CT 证实左大脑中动脉区有一梗塞灶。脑动脉造影示左大脑中动脉完全闭塞。尿比重升高,蛋白卅,24小时尿蛋白11.4(正常<0.1)g。血纤维蛋白原升高至70l(正常160～340)mg/dl,血纤溶酶原升高至145%(正常90%～110%),游离蛋白S 减少至55%(正常60%～140%),临床诊断为肾病综合征引起高凝状态,是继发于膜增殖性肾小球肾炎。使用肝素、华法令,症状好转。
The authors report 2 adult acute cerebral infarction, all suffering from nephrotic syndrome caused by hypercoagulable state. Example 1 male, 36 years old. Due to weakness of the right limb, can not speak to hospital. Check with hemianopia, right lower body feel decreased. CT confirmed that there is a infarction in the left middle cerebral artery area. Cerebral arteriography showed complete occlusion of the left middle cerebral artery. Urinary specific gravity increased, protein 卅, 24-hour urinary protein 11.4 (normal <0.1) g. Fibrinogen increased to 70 l (normal 160 to 340) mg / dl, plasminogen increased to 145% (normal 90% to 110%), and free protein S reduced to 55% (normal 60% to 140 %), The clinical diagnosis of nephrotic syndrome caused by hypercoagulable state, is secondary to membranoproliferative glomerulonephritis. Heparin, warfarin, the symptoms improved.