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多年以来，许多学者一直认为，癌症与衰老是在细胞调亡（apoptosis）调节机制的控制之下。现在，关于端粒和瑞粒酶的研究有可能改变这一观点：即细胞癌变与衰老主要是受细胞染色体两端的重复序列一端粒（tel－mere）长度的控制，端粒随每一次的细胞分裂而缩短，当缩短到临界长
For many years, many scholars have been convinced that cancer and aging are under the control of apoptosis regulation mechanism. Now, studies on telomeres and chymase may change this notion that cell canceration and senescence are mainly controlled by the tel-mere length of the repeats on both ends of the cell’s chromosomes. Telomeres follow each cell. Split and shorten, when shortened to critical length