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结肠腺瘤样息肉(APC)肿瘤抑制基因的突变是引发85%的结肠癌病例的主要原因。目前,来自于美国犹他州立大学卫生科学中心的研究人员们弄清了其中的原因,相关结果已发表在最新的《生物化学杂志》的网络版上。研究人员对此解释说,APC可控制饮食中的维生素 A 转变为视黄酸的过程;一旦该过程受损
Mutations in tumor suppressor genes of the colon adenomatous polyps (APC) are the major cause of 85% of colon cancer cases. Researchers at the Center for Health Sciences at Utah State University have now figured out why, and the results have been published in the latest online edition of the journal Biochemistry. The researchers explained that APC can control dietary vitamin A into retinoic acid process; once the process is damaged