应激是机体的非特异性保护适应机制 ,但它也引起机体自稳态的变动 ,甚至导致疾病。最近的研究显示 ,应激通过神经内分泌、免疫、氧化作用的介导对脂肪细胞自身及分泌、代谢等的影响 ,而导致胰岛素抵抗。干预应激对脂肪细胞的影响 ,可改善胰岛素抵抗 Stress is the body’s non-specific protective adaptation mechanism, but it also causes the body’s self-steady state changes, and even lead to disease. Recent studies have shown that stress induces insulin resistance through mediation of neuroendocrine, immune, and oxidative effects on adipocytes themselves and their secretion, metabolism, and the like. Intervention of stress on fat cells can improve insulin resistance